Non-thrombotic occlusion of the cavernous carotid from subdurai empyema

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Abstract
Scattered reports of the significant angiographic findings in intracranial bacterial infection have demonstrated several types of vascular changes, many of which are non-specific. These include narrowing or spasm of the major vessels at the base of the brain, segmental arterial dilatation, peripheral arterial occlusions, aneurysmal formation due to vessel wall involvement by infection, arterio-venous shunting and a thickened vascular wall of the dura (Ferris et al., 1968). The angiographic appearance of narrowing of a vessel may result from cellular infiltration, oedema of the wall, extrinsic compression or irritability of the vessel wall with actual vasospasm (Ferris and Levine, 1973). Progression of these changes to non-thrombotic occlusion has been observed in the smaller arteries of the brain. This is the first case demonstrating radiographic and pathologic documentation of non-thrombotic occlusion of the internal carotid artery secondary to intracranial infection.

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