The Protective Role of HSP90 against 3-Hydroxykynurenine-Induced Neuronal Apoptosis
- 1 June 2001
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 284 (2) , 261-267
- https://doi.org/10.1006/bbrc.2001.4938
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- C-terminal regions of Hsp90 are important for trapping the nucleotide during the ATPase cycle 1 1Edited by R. HuberJournal of Molecular Biology, 2000
- CARDIOTROPHIN-1 REDUCES STRESS-INDUCED HEAT SHOCK PROTEIN PRODUCTION IN CARDIAC MYOCYTESCytokine, 2000
- The Highly Inducible Member of the 70 kDa Family of Heat Shock Proteins Increases Canine Distemper Virus Polymerase ActivityJournal of General Virology, 1996
- 3-Hydroxykynurenine Toxicity on the Rat Striatum In VivoThe Japanese Journal of Pharmacology, 1996
- Increased Concentrations of the Neurotoxin 3‐Hydroxykynurenine in the Frontal Cortex of HIV‐1‐Positive PatientsJournal of Neurochemistry, 1995
- Heat Shock Proteins hsp90 and hsp70 Protect Neuronal Cells from Thermal Stress but Not from Programmed Cell DeathJournal of Neurochemistry, 1994
- Assisting spontaneity: the role of Hsp90 and small Hsps as molecular chaperonesTrends in Biochemical Sciences, 1994
- INCREASED BRAIN 3-HYDROXYKYNURENINE IN HUNTINGTON'S DISEASEThe Lancet, 1989
- THE HEAT-SHOCK PROTEINSAnnual Review of Genetics, 1988
- Quinolinic Acid: An Endogenous Metabolite That Produces Axon-Sparing Lesions in Rat BrainScience, 1983