Blood flow compensates oxygen demand in the vulnerable ca3 region of the hippocampus during kainate-induced seizures
- 1 December 1984
- journal article
- research article
- Published by Elsevier in Neuroscience
- Vol. 13 (4) , 1039-1049
- https://doi.org/10.1016/0306-4522(84)90287-2
Abstract
No abstract availableThis publication has 39 references indexed in Scilit:
- Limbic seizure and brain damage produced by kainic acid: Mechanisms and relevance to human temporal lobe epilepsyPublished by Elsevier ,2003
- Autoradiographic visualization of [3H]kainic acid receptor subtypes in the rat hippocampusNeuroscience Letters, 1983
- Metabolic Changes in Cerebral Cortex, Hippocampus, and Cerebellum During Sustained Bicuculline‐Induced SeizuresJournal of Neurochemistry, 1981
- Electrographic, clinical and pathological alterations following systemic administration of kainic acid, bicuculline or pentetrazole: Metabolic mapping using the deoxyglucose method with special reference to the pathology of epilepsyNeuroscience, 1981
- The role of epileptic activity in hippocampal and ‘remote’ cerebral lesions induced by kainic acidBrain Research, 1980
- Regional cerebral blood flow in the rat during prolonged seizure activityBrain Research, 1980
- Epileptic Brain Damage: The Role of Systemic Factors That Modify Cerebral Energy MetabolismBrain, 1978
- CEREBRAL METABOLIC CHANGES DURING PROLONGED EPILEPTIC SEIZURES IN RATSJournal of Neurochemistry, 1977
- Cerebral pO2, pCO2 and pH: Changes During Convulsive Activity and their Significance for Spontaneous Arrest of SeizuresEpilepsia, 1972
- Two methods for selective silver impregnation of degenerating axons and their synaptic endings in the central nervous systemBrain Research, 1967