Abnormal Pattern of Tie-2 and Vascular Endothelial Growth Factor Receptor Expression in Human Cerebral Arteriovenous Malformations
- 1 October 2000
- journal article
- research article
- Published by Wolters Kluwer Health in Neurosurgery
- Vol. 47 (4) , 910-919
- https://doi.org/10.1097/00006123-200010000-00022
Abstract
OBJECTIVE. Human cerebral arteriovenous malformations (AVMs) are speculated to result from abnormal angiogenesis. Vascular endothelial growth factor reKeywords
This publication has 32 references indexed in Scilit:
- Molecular mediators of tumor angiogenesis: Enhanced expression and activation of vascular endothelial growth factor receptor KDR in primary breast cancerInternational Journal of Cancer, 1999
- Arteriovenous Malformations of the Brain in AdultsNew England Journal of Medicine, 1999
- Spontaneous Angiographic Obliteration of Cerebral Arteriovenous MalformationsNeurosurgery, 1999
- Redefined role of angiogenesis in the pathogenesis of dural arteriovenous malformationsJournal of Neurosurgery, 1997
- Signaling Vascular Morphogenesis and MaintenanceScience, 1997
- Blood Vessel Formation: What Is Its Molecular Basis?Cell, 1996
- Tie Endothelial Cell-specific Receptor Tyrosine Kinase Is Upregulated in the Vasculature of Arteriovenous MalformationsJournal of Neuropathology and Experimental Neurology, 1996
- Role of the Flt-1 receptor tyrosine kinase in regulating the assembly of vascular endotheliumNature, 1995
- Dominant-negative and targeted null mutations in the endothelial receptor tyrosine kinase, tek, reveal a critical role in vasculogenesis of the embryo.Genes & Development, 1994
- The related FLT4, FLT1, and KDR receptor tyrosine kinases show distinct expression patterns in human fetal endothelial cells.The Journal of Experimental Medicine, 1993