Modulation of MAPK pathways and cell cycle by replicating hepatitis B virus: Factors contributing to hepatocarcinogenesis
- 30 September 2007
- journal article
- Published by Elsevier in Journal of Hepatology
- Vol. 47 (3) , 325-337
- https://doi.org/10.1016/j.jhep.2007.03.025
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Activation of extracellular signal‐regulated kinase (ERK) in G2 phase delays mitotic entry through p21CIP1Cell Proliferation, 2006
- Activation of Focal Adhesion Kinase by Hepatitis B Virus HBx Protein: Multiple Functions in Viral ReplicationJournal of Virology, 2006
- The influence of the signal dynamics of activated form of IKK on NF‐κB and anti‐apoptotic gene expressions: A systems biology approachFEBS Letters, 2006
- Induced oxidative stress and activated expression of manganese superoxide dismutase during hepatitis C virus replication: role of JNK, p38 MAPK and AP-1Biochemical Journal, 2004
- p38 Mitogen-Activated Protein Kinase Mediates Cell Death and p21-Activated Kinase Mediates Cell Survival during Chemotherapeutic Drug-induced Mitotic ArrestMolecular Biology of the Cell, 2003
- PI3K-FRAP/mTOR pathway is critical for hepatocyte proliferation whereas MEK/ERK supports both proliferation and survivalHepatology, 2002
- Hepatitis B Virus X Protein Differentially Activates RAS-RAF-MAPK and JNK Pathways in X-transforming Versus Non-transforming AML12 HepatocytesJournal of Biological Chemistry, 2001
- Role of NF-κB and Myc Proteins in Apoptosis Induced by Hepatitis B Virus HBx ProteinJournal of Virology, 2001
- Activators and target genes of Rel/NF-κB transcription factorsOncogene, 1999
- Cell cycle arrest and DNA endoreduplication following p21Waf1/Cip1 expressionOncogene, 1998