QT interval prolongation by non-cardiovascular drugs: issues and solutions for novel drug development
- 1 July 1999
- journal article
- Published by Elsevier in Pharmaceutical Science & Technology Today
- Vol. 2 (7) , 270-280
- https://doi.org/10.1016/s1461-5347(99)00172-8
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Potential Molecular Basis of Different Physiological Properties of the Transient Outward K + Current in Rabbit and Human Atrial MyocytesCirculation Research, 1999
- The Molecular Ggenetics of the Long QT Syndrome: Genes Causing Fainting and Sudden DeathAnnual Review of Medicine, 1998
- Decreased Density of Ito in Left Ventricular Myocytes from German Shepherd Dogs with Inherited ArrhythmiasJournal of Cardiovascular Electrophysiology, 1997
- Shal‐type channels contribute to the Ca2+‐independent transient outward K+ current in rat ventricle.The Journal of Physiology, 1997
- Electrophysiological and pharmacological correspondence between Kv4.2 current and rat cardiac transient outward currentCardiovascular Research, 1997
- Role of the Kv4.3 K + Channel in Ventricular MuscleCirculation Research, 1996
- Description of a Nonselective Cation Current in Human AtriumCirculation Research, 1995
- Drug-Induced Torsade de PointesDrug Safety, 1994
- Quantitative analysis of potassium channel mRNA expression in atrial and ventricular muscle of rats.Circulation Research, 1994
- Influence of Tedisamil on the Initiation and Maintenance of Ventricular FibrillationJournal of Cardiovascular Pharmacology, 1991