The effect of 1H-[1,2,4]oxadiazolo[4,3-a]quinoxalin-1-one (ODQ) and charybdotoxin (CTX) on relaxations of isolated cerebral arteries to nitric oxide

This publication has 28 references indexed in Scilit:

• Bright and dark sides of nitric oxide in ischemic brain injury
  Trends in Neurosciences, 1997
• Nitric oxide and cGMP cause vasorelaxation by activation of a charybdotoxin-sensitive K channel by cGMP-dependent protein kinase.
  Proceedings of the National Academy of Sciences, 1994
• NITRIC OXIDE: A Physiologic Messenger Molecule
  Annual Review of Biochemistry, 1994
• Nitric oxide directly activates calcium-dependent potassium channels in vascular smooth muscle
  Nature, 1994
• Nitric oxide and the cerebral circulation.
  Stroke, 1994
• Zaprinast increases cyclic GMP levels in plasma and in aortic tissue of rats
  European Journal of Pharmacology, 1993
• Purification, sequence, and model structure of charybdotoxin, a potent selective inhibitor of calcium-activated potassium channels.
  Proceedings of the National Academy of Sciences, 1988
• The Pharmacological and Physiological Role of Cyclic GMP in Vascular Smooth Muscle Relaxation
  Annual Review of Pharmacology and Toxicology, 1985
• Vasopressin causes endothelium-dependent relaxations of the canine basilar artery.
  Circulation Research, 1984
• The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine
  Nature, 1980