Differential inactivation of inotropic and toxic digitalis receptors in ischemic dog heart. Molecular basis of the deleterious effects of digitalis.

This publication has 22 references indexed in Scilit:

• Early phase acute myocardial infarct size quantification: Validation of the triphenyl tetrazolium chloride tissue enzyme staining technique
  Published by Elsevier ,2004
• Myocardial ischemia revisited. The osmolar load, membrane damage, and reperfusion
  Journal of Molecular and Cellular Cardiology, 1986
• Association of digitalis therapy with mortality in survivors of acute myocardial infarction: Observations in the beta-blocker heart attack trial
  Journal of the American College of Cardiology, 1985
• Limitations of fluorocarbons in reducing myocardial infarct size
  The American Journal of Cardiology, 1985
• Enhancement of triggered activity in ischemic purkinje fibers by ouabain: A mechanism of increased susceptibility to digitalis toxicity in myocardial infarction
  Journal of the American College of Cardiology, 1985
• The effect of lipid intermediates on Ca2+ and Na+ permeability and (Na+ + K+)-ATPase of cardiac sarcolemma. A possible role in myocardial ischemia
  Biochimica et Biophysica Acta (BBA) - Biomembranes, 1984
• Ca2+-free perfusion of rat heart reveals a (Na+ + K+) ATPase form highly sensitive to ouabain
  Nature, 1982
• Precordial electrocardiographic mapping. A technique to assess the efficacy of interventions designed to limit infarct size.
  Circulation, 1978
• Ischemia-induced alterations in myocardial (Na+ + K+)-ATPase and cardiac glycoside binding.
  Journal of Clinical Investigation, 1976
• Accelerated cardiac escape rhythms caused by ouabain intoxication
  The American Journal of Cardiology, 1974