Matrix Protein Mutations Contribute to Inefficient Induction of Apoptosis Leading to Persistent Infection of Human Neural Cells by Vesicular Stomatitis Virus
- 1 March 2002
- Vol. 295 (1) , 63-73
- https://doi.org/10.1006/viro.2001.1329
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- Matrix Protein and Another Viral Component Contribute to Induction of Apoptosis in Cells Infected with Vesicular Stomatitis VirusJournal of Virology, 2001
- Oncolytic Activity of Vesicular Stomatitis Virus Is Effective against Tumors Exhibiting Aberrant p53, Ras, or Myc Function and Involves the Induction of ApoptosisJournal of Virology, 2001
- Mammalian Caspases: Structure, Activation, Substrates, and Functions During ApoptosisAnnual Review of Biochemistry, 1999
- Virus infection induces neuronal apoptosis: A comparison with trophic factor withdrawalCell Death & Differentiation, 1998
- Pathogenesis of mouse hepatitis virus-induced demyelinationJournal of NeuroVirology, 1996
- Induction of apoptotic DNA fragmentation by the infection of vesicular stomatitis virusVirus Research, 1995
- Viruses and apoptosisCurrent Opinion in Genetics & Development, 1995
- Viral persistenceCell, 1989
- Vesicular Stomatitis Virus Can Establish Persistent Infections in Syrian HamstersJournal of General Virology, 1982
- On the Mechanism of Neurotropism of Vesicular Stomatitis Virus in Newborn Hamsters. Studies with Temperature-sensitive MutantsJournal of General Virology, 1975