PARALYSIS OF CRANIAL NERVES COMPLICATING HERPES ZOSTER

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Abstract
HERPES zoster is presumed to be caused by a filtrable virus similar to, if not identical with, that causing varicella. This virus is capable of producing an acute inflammatory process in cranial or spinal nerves, ganglions, dorsal and ventral spinal nerve roots, posterior gray matter of the spinal cord or the adjacent meninges, and the disease may therefore be appropriately termed a neuroganglioradiculomeningomyelitis. Clinical manifestations consist of a cutaneous grouped vesicular eruption on an erythematous base, neuralgia and, occasionally, paralysis. Many predisposing factors have been suggested as favoring the establishment of herpes zoster: seasonal variations; trauma; arsenical drugs; dental pathologic changes; spondylitis, dyscrasias of the blood, including leukemia, Hodgkin's disease and lymphosarcoma; malignant visceral tumors, and tabes dorsalis. Sex is not a factor, and herpes zoster has been reported in all age groups, although it occurs most frequently in middle-aged or elderly persons. Statistical proof that herpes zoster

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