Involvement of Oxidation-Sensitive Mechanisms in the Cardiovascular Effects of Hypercholesterolemia

This publication has 173 references indexed in Scilit:

• Lipophilic HMG-CoA reductase inhibitor has an anti-inflammatory effect: Reduction of MRNA levels for interleukin-1β, interleukin-6, cyclooxygenase-2, and p22phox by regulation of peroxisome proliferator-activated receptor α (PPARα) in primary endothelial cells
  Life Sciences, 2000
• Coronary Endothelial Function Is Preserved With Chronic Endothelin Receptor Antagonism in Experimental Hypercholesterolemia In Vitro
  Arteriosclerosis, Thrombosis, and Vascular Biology, 1999
• Prostaglandin F₂-Like Compounds, F₂-Isoprostanes, Are Present in Increased Amounts in Human Atherosclerotic Lesions
  Arteriosclerosis, Thrombosis, and Vascular Biology, 1997
• Activation of Nuclear Factor-κB in Cultured Endothelial Cells by Increased Glucose Concentration: Prevention by Calphostin C
  Journal of Cardiovascular Pharmacology, 1997
• Enhanced Expression of Endothelial Oxidized Low-Density Lipoprotein Receptor (LOX-1) in Hypertensive Rats
  Biochemical and Biophysical Research Communications, 1997
• The potential use of angiotensin-converting enzyme inhibitors in patients with hyperlipidemia
  The American Journal of Cardiology, 1997
• Physiologic consequences of increased vascular oxidant stresses in hypercholesterolemia and atherosclerosis: Implications for impaired vasomotion
  The American Journal of Cardiology, 1995
• The pathogenesis of atherosclerosis: a perspective for the 1990s
  Nature, 1993
• Hypercholesterolemia causes generalized impairment of endothelium-dependent relaxation to aggregating platelets in porcine arteries
  Journal of the American College of Cardiology, 1989
• Paradoxical Vasoconstriction Induced by Acetylcholine in Atherosclerotic Coronary Arteries
  New England Journal of Medicine, 1986