The fifth component of complement (C5) is necessary for maximal pulmonary leukocytosis in mice chronically exposed to cigarette smoke
- 1 April 1987
- journal article
- research article
- Published by Elsevier in Clinical Immunology and Immunopathology
- Vol. 43 (1) , 73-81
- https://doi.org/10.1016/0090-1229(87)90158-9
Abstract
No abstract availableThis publication has 22 references indexed in Scilit:
- Cigarette smoke can activate the alternative pathway of complement in vitro by modifying the third component of complement.Journal of Clinical Investigation, 1985
- Nicotine is Chemotactic for Neutrophils and Enhances Neutrophil Responsiveness to Chemotactic PeptidesScience, 1984
- Expression of C5a-like biological activities by the fifth component of human complement (C5) upon limited digestion with noncomplement enzymes without release of polypeptide fragments.The Journal of Experimental Medicine, 1983
- Chemotactic activity of elastin-derived peptides.Journal of Clinical Investigation, 1980
- Human Alveolar Macrophage-derived Chemotactic Factor for NeutrophilsJournal of Clinical Investigation, 1980
- Alveolar macrophage-derived chemotactic factor: kinetics of in vitro production and partial characterization.Journal of Clinical Investigation, 1980
- Mechanism for the inflammatory response in primate lungs. Demonstration and partial characterization of an alveolar macrophage-derived chemotactic factor with preferential activity for polymorphonuclear leukocytes.Journal of Clinical Investigation, 1977
- Collagen-and collagen peptide-induced chemotaxis of human blood monocytes.The Journal of Experimental Medicine, 1976
- Leukocyte Recruitment to Airways by Cigarette Smoke and Particle Phase in Contrast to Cytotoxicity of VaporScience, 1975
- LEUKOCYTE LOCOMOTION AND CHEMOTAXISThe Journal of Experimental Medicine, 1973