CTLA-4 Ligation Suppresses CD28-induced NF-κB and AP-1 Activity in Mouse T Cell Blasts
Open Access
- 1 May 1999
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 274 (20) , 14400-14405
- https://doi.org/10.1074/jbc.274.20.14400
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- NF-κB AND REL PROTEINS: Evolutionarily Conserved Mediators of Immune ResponsesAnnual Review of Immunology, 1998
- THE NF-κB AND IκB PROTEINS: New Discoveries and InsightsAnnual Review of Immunology, 1996
- CD28/B7 SYSTEM OF T CELL COSTIMULATIONAnnual Review of Immunology, 1996
- T CELL ANTIGEN RECEPTOR SIGNAL TRANSDUCTION PATHWAYSAnnual Review of Immunology, 1996
- Superantigen responses and co-stimulation: CD28 and CTLA-4 have opposing effects on T cell expansion in vitro and in vivoInternational Immunology, 1996
- Lymphoproliferative Disorders with Early Lethality in Mice Deficient in Ctla-4Science, 1995
- Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4Immunity, 1995
- CD28 and CTLA-4 have opposing effects on the response of T cells to stimulation.The Journal of Experimental Medicine, 1995
- CTLA-4 can function as a negative regulator of T cell activationImmunity, 1994
- Coexpression and functional cooperation of CTLA-4 and CD28 on activated T lymphocytes.The Journal of Experimental Medicine, 1992