Alpha-Phenyl-tert-butylnitrone (PBN) inhibits NFκB activation offering protection against chemically induced diabetes
- 15 February 2000
- journal article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 28 (4) , 604-614
- https://doi.org/10.1016/s0891-5849(99)00271-3
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Low antioxidant enzyme gene expression in pancreatic islets compared with various other mouse tissuesPublished by Elsevier ,1999
- Nuclear Factor Kappa B: Important Transcription Factor and Therapeutic TargetThe Journal of Clinical Pharmacology, 1998
- Generation of Nitric Oxide from Streptozotocin (STZ) in the Presence of Copper(II) plus Ascorbate: Implication for the Development of STZ-Induced DiabetesBiochemical and Biophysical Research Communications, 1998
- Oxidants, Transcription Factors, and Intestinal InflammationJournal of Clinical Gastroenterology, 1997
- THE NF-κB AND IκB PROTEINS: New Discoveries and InsightsAnnual Review of Immunology, 1996
- Structure, Regulation and Function of NF-kappaBAnnual Review of Cell Biology, 1994
- Protection against oxidative damage to CNS by α-phenyl-tert-butyl nitrone (PBN) and other spin-trapping agents: A novel series of nonlipid free radical scavengersJournal of Molecular Neuroscience, 1991
- Conference summary: Diet as an environmental factor in development of insulin-dependent diabetes mellitusCanadian Journal of Physiology and Pharmacology, 1991
- Alloxan-induced free radical production in isolated cells. Selective effect on islet cellsDiabetes, 1984
- DNA strand breaks in pancreatic islets by administration of alloxan or streptozotocinBiochemical and Biophysical Research Communications, 1981