Natural Killer Cells Are Polarized Toward Cytotoxicity in Chronic Hepatitis C in an Interferon-Alfa–Dependent Manner
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Open Access
- 1 January 2010
- journal article
- Published by Elsevier in Gastroenterology
- Vol. 138 (1) , 325-335.e2
- https://doi.org/10.1053/j.gastro.2009.08.066
Abstract
No abstract availableKeywords
Funding Information
- National Institute of Diabetes and Digestive and Kidney Diseases
- Deutsche Forschungsgemeinschaft
- National Institutes of Health (AH173/1-1)
This publication has 43 references indexed in Scilit:
- Natural killer cell function is intact after direct exposure to infectious hepatitis C virions†Hepatology, 2008
- Natural killer cells suppress full cycle HCV infection of human hepatocytesJournal of Viral Hepatitis, 2008
- Distinct KIR/HLA compound genotypes affect the kinetics of human antiviral natural killer cell responsesJournal of Clinical Investigation, 2008
- Hepatic gene expression during treatment with peginterferon and ribavirin: Identifying molecular pathways for treatment responseHepatology, 2007
- High basal STAT4 balanced by STAT1 induction to control type 1 interferon effects in natural killer cellsThe Journal of Experimental Medicine, 2007
- Liver-Infiltrating Lymphocytes in Chronic Human Hepatitis C Virus Infection Display an Exhausted Phenotype with High Levels of PD-1 and Low Levels of CD127 ExpressionJournal of Virology, 2007
- Cytokines induced during chronic hepatitis B virus infection promote a pathway for NK cell–mediated liver damageThe Journal of Experimental Medicine, 2007
- Virus-induced type I IFN stimulates generation of immunoproteasomes at the site of infectionJournal of Clinical Investigation, 2006
- Tumour necrosis factor related apoptosis inducing ligand (TRAIL) induces hepatic steatosis in viral hepatitis and after alcohol intakeGut, 2005
- Immunology of hepatitis B virus and hepatitis C virus infectionNature Reviews Immunology, 2005