Nafazatrom, an arachidonate metabolism inhibitor, decreases prolactin and GH release

This publication has 16 references indexed in Scilit:

• Angiotensin Peptides Stimulate Phosphoinositide Breakdown and Prolactin Release in Anterior Pituitary Cells in Culture*
  Endocrinology, 1986
• Maitotoxin, a Calcium Channel Activator, Increases Prolactin Release from Rat Pituitary Tumor 7315a Cells by a Mechanism That May Involve Leukotriene Production
  Neuroendocrinology, 1986
• 5-Hydroxyeicosatetraenoic Acid Increases Prolactin Release from Rat Anterior Pituitary Cells*
  Endocrinology, 1985
• Comparison of the effects of some compounds on human neutrophil degranulation and leukotriene B4 and thromboxane B2 synthesis
  Biochemical Pharmacology, 1985
• Evidence Supporting a Correlation between Arachidonic Acid Release and Prolactin Secretion from GH₃Cells*
  Endocrinology, 1985
• The Effects of Maitotoxin on⁴⁵Ca²⁺Flux and Hormone Release in GH3 Rat Pituitary Cells*
  Endocrinology, 1985
• Arachidonate Stimulates Prolactin Releasein Vitro: A Role for the Fatty Acid and Its Metabolites as Intracellular Regulator(s) in Mammotrophs*
  Endocrinology, 1985
• Modification of Basal and GRF-Stimulated Cyclic AMP Levels and Growth Hormone Release by Phospholipid Metabolic Enzyme Inhibitors
  Neuroendocrinology, 1985
• A Possible Role of Prostacyclin to Stimulate Prolactin and Growth Hormone Release by Hypothalamic and Pituitary Actions, Respectively*
  Endocrinology, 1984
• Nafazatrom (bay g 6575) inhibition of tumor cell lipoxygenase activity and cellular proliferation
  FEBS Letters, 1982