Imipramine and phenelzine decrease glutamate overflow in the prefrontal cortex—a possible mechanism of neuroprotection in major depression?
- 1 October 2000
- journal article
- research article
- Published by Elsevier in Neuroscience
- Vol. 100 (4) , 681-684
- https://doi.org/10.1016/s0306-4522(00)00390-0
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Neural plasticity to stress and antidepressant treatmentPublished by Elsevier ,1999
- Acute application of the tricyclic antidepressant desipramine presynaptically stimulates the exocytosis of glutamate in the hippocampusNeuroscience, 1999
- Temporal dynamics of glutamate efflux in the prefrontal cortex and in the hippocampus following repeated stress: effects of pretreatment with saline or diazepamPublished by Elsevier ,1998
- Glutamate release in human cerebral cortex and its modulation by 5‐hydroxtryptamine acting at h 5‐HT1D receptorsBritish Journal of Pharmacology, 1998
- Possible mechanisms for atrophy of the human hippocampusMolecular Psychiatry, 1997
- Adaptation of cortical but not hippocampal NMDA receptors after chronic citalopram treatmentEuropean Journal of Pharmacology, 1996
- Antidepressant-like actions of the polyamine site NMDA antagonist, eliprodil (SL-82.0715)Pharmacology Biochemistry and Behavior, 1995
- Stress Preferentially Increases Extraneuronal Levels of Excitatory Amino Acids in the Prefrontal Cortex: Comparison to Hippocampus and Basal GangliaJournal of Neurochemistry, 1993
- Glutamate neurotoxicity and diseases of the nervous systemNeuron, 1988
- Inhibitory effects of noradrenaline and dopamine on calcium influx and neurotransmitter glutamate release in mammalian brain slicesEuropean Journal of Pharmacology, 1987