An Akt/β-Arrestin 2/PP2A Signaling Complex Mediates Dopaminergic Neurotransmission and Behavior
Top Cited Papers
- 1 July 2005
- Vol. 122 (2) , 261-273
- https://doi.org/10.1016/j.cell.2005.05.012
Abstract
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This publication has 50 references indexed in Scilit:
- Transduction of Receptor Signals by ß-ArrestinsScience, 2005
- Protein Phosphatase 2A Negatively Regulates Insulin's Metabolic Signaling Pathway by Inhibiting Akt (Protein Kinase B) Activity in 3T3-L1 AdipocytesMolecular and Cellular Biology, 2004
- The effects of clozapine on the GSK‐3‐mediated signaling pathwayFEBS Letters, 2004
- Topographical Assessment of Ethological and Dopamine Receptor Agonist-Induced Behavioral Phenotype in Mutants with Congenic DARPP-32 ‘Knockout’Neuropsychopharmacology, 2003
- α-Thrombin Induces Rapid and Sustained Akt Phosphorylation by β-Arrestin1-dependent and -independent Mechanisms, and Only the Sustained Akt Phosphorylation Is Essential for G1 Phase ProgressionJournal of Biological Chemistry, 2002
- A Paradigm Shift in Brain ResearchScience, 2001
- PKB/AKT: functional insights from genetic modelsNature Reviews Molecular Cell Biology, 2001
- β-Arrestin-Dependent Formation of β 2 Adrenergic Receptor-Src Protein Kinase ComplexesScience, 1999
- AR-A014418, a selective GSK-3 inhibitor, produces antidepressant-like effects in the forced swim testInternational Journal of Neuropsychopharmacology, 1999
- Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase BNature, 1995