Abstract
Experiments on the uptake of acriflavine by uninfected E. coli cells and by cells infected with acridine-sensitive or -resistant bacteriophage show that (1) sensitivity to acriflavine is related to a greatly increased uptake of dye by infected cells; (2) one of the two mutations to acridine resistance in bacteriophage T2 and T4 results in a reduced uptake of dye; and (3) the process responsible for increased acriflavine uptake is dependent on phage-direct protein synthesis in the infected cells.