Activation of the Oxidative Stress Pathway by HIV-1 Vpr Leads to Induction of Hypoxia-inducible Factor 1α Expression

This publication has 59 references indexed in Scilit:

• A Potential α-Helix Motif in the Amino Terminus of LANA Encoded by Kaposi's Sarcoma-Associated Herpesvirus Is Critical for Nuclear Accumulation of HIF-1α in Normoxia
  Journal of Virology, 2007
• Activation of hypoxia-inducible factor 1 in human T-cell leukaemia virus type 1-infected cell lines and primary adult T-cell leukaemia cells
  Biochemical Journal, 2007
• Novel Nuclear Import of Vpr Promoted by Importin α Is Crucial for Human Immunodeficiency Virus Type 1 Replication in Macrophages
  Journal of Virology, 2007
• Hypoxia-inducible factor 1α stabilization by carbon monoxide results in cytoprotective preconditioning
  Proceedings of the National Academy of Sciences, 2007
• Pathological apoptosis in the developing brain
  Apoptosis, 2007
• Inhibition of wild-type p66ShcA in mesangial cells prevents glycooxidant-dependent FOXO3a regulation and promotes the survival phenotype
  American Journal of Physiology-Renal Physiology, 2007
• Hypoxia-inducible factors: central regulators of the tumor phenotype
  Current Opinion in Genetics & Development, 2007
• The signaling mechanism of ROS in tumor progression
  Cancer and Metastasis Reviews, 2006
• HIV-associated dementia, mitochondrial dysfunction, and oxidative stress
  Mitochondrion, 2004
• HIF1A Gene Transcription Is Dependent on a Core Promoter Sequence Encompassing Activating and Inhibiting Sequences Located Upstream from the Transcription Initiation Site and cis Elements Located within the 5′UTR
  Biochemical and Biophysical Research Communications, 1999