Ischemic Preconditioning Increases iNOS Transcript Levels in Conscious Rabbits via a Nitric Oxide-dependent Mechanism
- 31 August 1999
- journal article
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 31 (8) , 1469-1481
- https://doi.org/10.1006/jmcc.1999.0983
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Non-Enzymatic Production of Nitric Oxide (NO) from NO Synthase InhibitorsBiochemical and Biophysical Research Communications, 1998
- Ablation of the murine alpha myosin heavy chain gene leads to dosage effects and functional deficits in the heart.Journal of Clinical Investigation, 1996
- Mechanisms of Interleukin-1β Regulation of Nitric Oxide Synthase in Cardiac MyocytesHypertension, 1996
- In SituIdentification of Messenger RNA of Endothelial Type Nitric Oxide Synthase in Rat Cardiac MyocytesBiochemical and Biophysical Research Communications, 1996
- Involvement of inducible nitric oxide synthase in the inflammatory process of myocardial infarctionInternational Journal of Cardiology, 1995
- Plasma Nitric Oxide End Products Are Increased in the Ischemic Canine HeartBiochemical and Biophysical Research Communications, 1995
- Late preconditioning against myocardial stunning. An endogenous protective mechanism that confers resistance to postischemic dysfunction 24 h after brief ischemia in conscious pigs.Journal of Clinical Investigation, 1995
- Inducible Nitric Oxide Synthase Activity in Myocardium After Myocardial Infarction in RabbitBiochemical and Biophysical Research Communications, 1994
- Dexamethasone Inhibits the Expression of an Inducible Nitric Oxide Synthase in Infarcted Rabbit MyocardiumBiochemical and Biophysical Research Communications, 1994
- Requirement for Transcription Factor IRF-1 in NO Synthase Induction in MacrophagesScience, 1994