The 9p-syndrome.

Abstract
A 13 yr old boy with 9p- (p22 .fwdarw. pter) is reported. He had many features in common with previous 9p- cases and several distinctive features including polydactyly and precocious puberty. Cytogenetic studies revealed a de novo deletion distal to band 9p22, which was the reported site of chromosome break in 9 of the 10 previous 9p- cases. Evaluation of the human GALT (galactose-1-phosphate uridyl transferase EC 2.7.7.12) enzyme suggests that its locus is not on the deleted segment.