Prostaglandin-concentration-dependent desensitization of adenylate cyclase in human erythroleukaemia (HEL) cells is abolished by pertussis toxin and enhanced by induction by dimethyl sulphoxide
- 15 December 1991
- journal article
- Published by Portland Press Ltd. in Biochemical Journal
- Vol. 280 (3) , 801-804
- https://doi.org/10.1042/bj2800801
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- NOVEL MECHANISM OF HETEROLOGOUS DESENSITIZATION OF ADENYLATE-CYCLASE - PROSTAGLANDINS BIND WITH DIFFERENT AFFINITIES TO BOTH STIMULATORY AND INHIBITORY RECEPTORS ON PLATELETS1990
- Expression of thromboxane A2 receptor in cultured human erythroleukemia cells and its induction by 12-O-tetradecanoylphorbol-13-acetateBiochemical and Biophysical Research Communications, 1989
- Cyclic AMP turnover in response to prostaglandins in intact platelets: evidence for separate stimulatory and inhibitory prostaglandin receptors.1988
- Effects of activation of protein kinase C on the agonist-induced stimulation and inhibition of cyclic AMP formation in intact human plateletsBiochemical Journal, 1987
- Protein kinase C interferes with Ni‐mediated inhibition of human platelet adenylate cyclaseFEBS Letters, 1985
- Modulation of adenylate cyclase of human platelets by phorbol esterEuropean Journal of Biochemistry, 1985
- [29] Receptors for PGI2 and PGD2 on human plateletsPublished by Elsevier ,1982
- Turnover of adenosine 3',5'-monophosphate in WI-38 cultured fibroblastsBiochemistry, 1980
- Adenylate cyclase assay.1979
- Pharmacology and endogenous roles of prostaglandin endoperoxides, thromboxane A2, and prostacyclin.1978