Specific Kinetic Alterations of Human CaV2.1 Calcium Channels Produced by Mutation S218L Causing Familial Hemiplegic Migraine and Delayed Cerebral Edema and Coma after Minor Head Trauma

This publication has 39 references indexed in Scilit:

• Familial Hemiplegic Migraine Type 1 Mutations K1336E, W1684R, and V1696I Alter Cav2.1 Ca2+ Channel Gating
  Journal of Biological Chemistry, 2004
• Presynaptic Ca2+ Channels Compete for Channel Type-Preferring Slots in Altered Neurotransmission Arising from Ca2+ Channelopathy
  Neuron, 2004
• A novel R1347Q mutation in the predicted voltage sensor segment of the P/Q‐type calcium‐channel α_1A‐subunit in a family with progressive cerebellar ataxia and hemiplegic migraine
  Clinical Genetics, 2003
• Neurobiology of migraine
  Nature Reviews Neuroscience, 2003
• Functional Consequences of P/Q-type Ca2+Channel Cav2.1 Missense Mutations Associated with Episodic Ataxia Type 2 and Progressive Ataxia
  Journal of Biological Chemistry, 2002
• Delayed cerebral edema and fatal coma after minor head trauma: Role of the CACNA1A calcium channel subunit gene and relationship with familial hemiplegic migraine
  Annals of Neurology, 2001
• Three New Familial Hemiplegic Migraine Mutants Affect P/Q-type Ca2+ Channel Kinetics
  Published by Elsevier ,2000
• Transfer of 1,4-Dihydropyridine Sensitivity from L-Type to Class A (BI) Calcium Channels
  Neuron, 1996
• Exocytotic Ca2+ channels in mammalian central neurons
  Trends in Neurosciences, 1995
• MIGRAINE COMA
  Brain, 1985