TNF-α and IFN-γ potentiate the deleterious effects of IL-1β on mouse pancreatic islets mainly via generation of nitric oxide
- 31 July 1994
- Vol. 6 (4) , 399-406
- https://doi.org/10.1016/1043-4666(94)90064-7
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Interleukin‐1 ‐induced expression of nitric oxide synthase in insulin‐producing cells is preceded by c‐fos induction and depends on gene transcription and protein synthesisFEBS Letters, 1993
- Analysis of gene expression: use of oligonucleotide primers for glyceraldehyde-3-phosphate dehydrogenase.Genome Research, 1992
- Interleukin-1β induces nitric oxide production and inhibits the activity of aconitase without decreasing glucose oxidation rates in isolated mouse pancreatic isletsBiochemical and Biophysical Research Communications, 1992
- Biochemical and Molecular Actions of Interleukin-1 on Pancreatic β-CellsAutoimmunity, 1991
- Interleukin-1β Induces an Early Decrease in Insulin Release, (Pro)Insulin Biosynthesis and Insulin Mrna in Mouse Pancreatic Islets by a Mechanism Dependent on Gene Transcription and Protein SynthesisAutoimmunity, 1991
- Inhibition of insulin secretion by interleukin‐1β and tumour necrosis factor‐α via an L‐arginine‐dependent nitric oxide generating mechanismFEBS Letters, 1990
- Interferon‐γ and tumor necrosis factor induce the L‐arginine‐dependent cytotoxic effector mechanism in murine macrophages*European Journal of Immunology, 1988
- Isolation of biologically active ribonucleic acid from sources enriched in ribonucleaseBiochemistry, 1979
- Isolated mouse pancreatic islets in culture: Effects of serum and different culture media on the insulin production of the isletsDiabetologia, 1978
- Untersuchungen uber die Harnstoffbildung im TierkörperHoppe-Seyler´s Zeitschrift Für Physiologische Chemie, 1932