The Met Receptor and α6β4 Integrin Can Function Independently to Promote Carcinoma Invasion
Open Access
- 1 July 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (31) , 32287-32293
- https://doi.org/10.1074/jbc.m403809200
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- The MSP Receptor Regulates α6β4 and α3β1 Integrins via 14-3-3 Proteins in Keratinocyte MigrationDevelopmental Cell, 2003
- NF-κB blockade and oncogenic Ras trigger invasive human epidermal neoplasiaNature, 2003
- Integrin (α6β4) regulation of eIF-4E activity and VEGF translationThe Journal of cell biology, 2002
- A Signaling Adapter Function for α6β4 Integrin in the Control of HGF-Dependent Invasive GrowthCell, 2001
- Identification of Insulin Receptor Substrate 1 (IRS-1) and IRS-2 as Signaling Intermediates in the α6β4 Integrin-Dependent Activation of Phosphoinositide 3-OH Kinase and Promotion of InvasionMolecular and Cellular Biology, 2001
- Cooperative Signaling between α6β4Integrin and ErbB-2 Receptor Is Required to Promote Phosphatidylinositol 3-Kinase-dependent InvasionPublished by Elsevier ,2000
- HGF induces FAK activation and integrin-mediated adhesion in MTLn3 breast carcinoma cellsInternational Journal of Cancer, 1999
- Structure and Function of Hemidesmosomes: More Than Simple Adhesion ComplexesJournal of Investigative Dermatology, 1999
- The integrin α6β4 and the biology of carcinomaBiochemistry and Cell Biology, 1996
- Beta4 integrin is required for hemidesmosome formation, cell adhesion and cell survival.The Journal of cell biology, 1996