EFFECTS OF LESIONS ON SUBCORTICALLY EVOKED MOVEMENT IN CAT

Abstract
Phasic movement of the contralateral forelimb was produced in cats by electrical stimulation of the caudate nucleus, hypothalamus, and anterior thalamus. Movement from caudate stimulation could be abolished by midline lesions at the junction of diencephalon and midbrain. These lesions did not seriously affect cortically induced movement. The results suggest a pathway for movement, distinct from the cortico-splnal tracts, that originates in or passes through the caudate nucleus, assumes a medial position through the diencephalon, then swings laterally in the midbrain to enter the basis pedunculi.

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