Inhibition of sodium‐dependent calcium overload to treat myocardial ischemia
Open Access
- 1 April 2006
- journal article
- research article
- Published by Wiley in Clinical Cardiology
- Vol. 29 (4) , 141-143
- https://doi.org/10.1002/clc.4960290403
Abstract
Click on the article title to read more.Keywords
This publication has 8 references indexed in Scilit:
- The mechanism of ranolazine action to reduce ischemia-induced diastolic dysfunctionEuropean Heart Journal Supplements, 2006
- Inhibition of late (sustained/ persistent) sodium current: a potential drug target to reduce intracellular sodium-dependent calcium overload and its detrimental effects on cardiomyocyte functionEuropean Heart Journal Supplements, 2004
- Electrophysiological Effects of Ranolazine, a Novel Antianginal Agent With Antiarrhythmic PropertiesCirculation, 2004
- Anti-ischemic effects and long-term survival during ranolazine monotherapy in patients with chronic severe anginaJournal of the American College of Cardiology, 2004
- 1068-128 Ranolazine inhibits late sodium current in isolated left ventricular myocytes of dogs with heart failureJournal of the American College of Cardiology, 2004
- Effects of Ranolazine With Atenolol, Amlodipine, or Diltiazem on Exercise Tolerance and Angina Frequency in Patients With Severe Chronic AnginaA Randomized Controlled TrialJAMA, 2004
- The Antianginal Agent Trimetazidine Does Not Exert Its Functional Benefit via Inhibition of Mitochondrial Long-Chain 3-Ketoacyl Coenzyme A ThiolaseCirculation Research, 2003
- Partial fatty acid oxidation (pFOX) inhibition: A new therapy for chronic stable anginaClinical Cardiology, 2003