Elevated levels of complement components C5 and C9 and decreased antitrypsin activity in the serum of patients with X-linked vacuolated myopathy
- 1 September 1996
- journal article
- research article
- Published by Wiley in Muscle & Nerve
- Vol. 19 (9) , 1144-1147
- https://doi.org/10.1002/(sici)1097-4598(199609)19:9<1144::aid-mus10>3.0.co;2-v
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- X‐linked vacuolated myopathy: Complement membrane attack complex on surface membrane of injured muscle fibersAnnals of Neurology, 1995
- Neurologic manifestations in Wegener's granulomatosis associated with α 1 ‐antitrypsin deficiencyNeurology, 1993
- IMMUNOCYTOCHEMICAL LOCALIZATION OF THE TERMINAL COMPLEMENT COMPLEX IN MULTIPLE SCLEROSISNeuropathology and Applied Neurobiology, 1989
- X‐Linked myopathy with excessive autophagy: A new hereditary muscle diseaseAnnals of Neurology, 1988
- Activation of terminal components of complement in patients with Guillain-Barré syndrome and other demyelinating neuropathies.Journal of Clinical Investigation, 1987
- Immunohistochemical Detection of the Membrane and Fluid‐phase Terminal Complement Complexes C5b‐9(m) and SC5b‐9Scandinavian Journal of Immunology, 1987
- Microvascular Deposition of Complement Membrane Attack Complex in DermatomyositisNew England Journal of Medicine, 1986
- Possible involvement of terminal complement complex in active Heymann nephritisKidney International, 1985
- Genes for immunoglobulin heavy chains and for α1-antitrypsin are localized to specific regions of chromosome 14qNature, 1982
- Alpha1-Antitrypsin DeficiencyNew England Journal of Medicine, 1978