In Vivo Chromatin Remodeling Events Leading to Inflammatory Gene Transcription under Diabetic Conditions
Open Access
- 1 April 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (17) , 18091-18097
- https://doi.org/10.1074/jbc.m311786200
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Translating the Histone CodeScience, 2001
- Histone AcetyltransferasesAnnual Review of Biochemistry, 2001
- Molecular Mechanisms of Tumor Necrosis Factor α Gene Expression in Monocytic Cells via Hyperglycemia-induced Oxidant Stress-dependent and -independent PathwaysJournal of Biological Chemistry, 2000
- RAGE Mediates a Novel Proinflammatory Axis: A Central Cell Surface Receptor for S100/Calgranulin PolypeptidesPublished by Elsevier ,1999
- Insufficient Glycemic Control Increases Nuclear Factor-κB Binding Activity in Peripheral Blood Mononuclear Cells Isolated From Patients With Type 1 DiabetesDiabetes Care, 1998
- Cellular receptors for advanced glycation end products. Implications for induction of oxidant stress and cellular dysfunction in the pathogenesis of vascular lesions.Arteriosclerosis and Thrombosis: A Journal of Vascular Biology, 1994
- The Effect of Glucose and Advanced Glycosylation End Products on IL‐6 Production by Human MonocytesAnnals of the New York Academy of Sciences, 1994
- Role of Oxidative Stress in Development of Complications in DiabetesDiabetes, 1991
- NF-κB: A pleiotropic mediator of inducible and tissue-specific gene controlCell, 1989
- Advanced Glycosylation End Products in Tissue and the Biochemical Basis of Diabetic ComplicationsNew England Journal of Medicine, 1988