Protein Kinase C Signaling Regulates ZO-1 Translocation and Increased Paracellular Flux of T84 Colonocytes Exposed toClostridium difficile Toxin A
Open Access
- 1 February 2002
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 277 (6) , 4247-4254
- https://doi.org/10.1074/jbc.m109254200
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Translocated EspF protein from enteropathogenic Escherichia coli disrupts host intestinal barrier functionJournal of Clinical Investigation, 2001
- Tight Junction, a Platform for Trafficking and Signaling Protein ComplexesThe Journal of cell biology, 2000
- Clostridium difficile toxin A causes early damage to mitochondria in cultured cellsGastroenterology, 2000
- p38 MAP kinase activation by Clostridium difficile toxin A mediates monocyte necrosis, IL-8 production, and enteritisJournal of Clinical Investigation, 2000
- Protein Interactions at the Tight JunctionJournal of Biological Chemistry, 1999
- Rho GTPasesJournal of Biological Chemistry, 1998
- Glucosylation and ADP Ribosylation of Rho Proteins: Effects on Nucleotide Binding, GTPase Activity, and Effector CouplingBiochemistry, 1998
- Glucosylation of Rho proteins by Clostridium difficile toxin BNature, 1995
- Clostridium difficile toxin B is more potent than toxin A in damaging human colonic epithelium in vitro.Journal of Clinical Investigation, 1995
- The low molecular mass GTP-binding protein Rho is affected by toxin A from Clostridium difficile.Journal of Clinical Investigation, 1995