Killing of Borrelia burgdorferi by macrophages is dependent on oxygen radicals and nitric oxide and can be enhanced by antibodies to outer surface proteins of the spirochete
- 13 November 2002
- journal article
- Published by Elsevier
- Vol. 40 (2) , 139-146
- https://doi.org/10.1016/0165-2478(94)90185-6
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Coiling Phagocytosis: A Way for MHC Class I Presentation of Bacterial Antigens?International Archives of Allergy and Immunology, 1994
- Protective immunity in lyme borreliosisParasitology Today, 1993
- Membrane sorting during phagocytosis: selective exclusion of major histocompatibility complex molecules but not complement receptor CR3 during conventional and coiling phagocytosis.The Journal of Experimental Medicine, 1992
- Nitric oxide: a pathogenetic factor in autoimmunityImmunology Today, 1992
- Experimental Borrelia burgdorferi infection in inbred mouse strains: Antibody response and association of H‐2 genes with resistance and susceptibility to development of arthritisEuropean Journal of Immunology, 1991
- A mouse model for Borrelia burgdorferi infection: approach to a vaccine against Lyme diseaseImmunology Today, 1991
- The role of nitrogen oxides as effector molecules of parasite killingParasitology Today, 1990
- The severe combined immunodeficiency (scid) mouse. A laboratory model for the analysis of Lyme arthritis and carditis.The Journal of Experimental Medicine, 1989
- Lyme DiseaseNew England Journal of Medicine, 1989
- Cell propagation on films of polymeric fluorocarbon as a means to regulate pericellular pH and pO2 in cultured monolayersFEBS Letters, 1971