Vasodilation to Bradykinin Is Mediated by an Ouabain-Sensitive Pathway as a Compensatory Mechanism for Impaired Nitric Oxide Availability in Essential Hypertensive Patients
- 28 September 1999
- journal article
- other
- Published by Wolters Kluwer Health in Circulation
- Vol. 100 (13) , 1400-1405
- https://doi.org/10.1161/01.cir.100.13.1400
Abstract
Background—In essential hypertension, endothelium-dependent vasodilation is impaired because of reduced nitric oxide (NO) availability, which is mainly caused by oxidative stress. The present study...Keywords
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