Possible involvement of spinal protein kinase C in thermal allodynia and hyperalgesia in diabetic mice
- 1 May 1999
- journal article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 372 (3) , 221-228
- https://doi.org/10.1016/s0014-2999(99)00228-9
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Nociceptive responses to high and low rates of noxious cutaneous heating are mediated by different nociceptors in the rat: behavioral evidencePain, 1996
- Contribution of protein kinase C to central sensitization and persistent pain following tissue injuryNeuroscience Letters, 1992
- Protein kinase C reduces Mg2+ block of NMDA-receptor channels as a mechanism of modulationNature, 1992
- Pretreatment with gangliosides reduces abnormal nociceptive responses associated with a rodent peripheral mononeuropathyPain, 1992
- Interactions between substance P, calcitonin gene-related peptide, taurine and excitatory amino acids in the spinal cordPain, 1990
- Streptozotocin and alloxan produce alterations in rat brain monoamines independently of pancreatic beta cells destructionLife Sciences, 1990
- Protein kinase C is activated in glomeruli from streptozotocin diabetic rats. Possible mediation by glucose.Journal of Clinical Investigation, 1989
- Calphostin C (UCN-1028C), a novel microbial compound, is a highly potent and specific inhibitor of protein kinase CBiochemical and Biophysical Research Communications, 1989
- Sorbitol, Phosphoinositides, and Sodium-Potassium-ATPase in the Pathogenesis of Diabetic ComplicationsNew England Journal of Medicine, 1987
- A study of pain threshold in diabetics with neuropathic foot lesions.Journal of Neurology, Neurosurgery & Psychiatry, 1986