DNA fragmentation during exposure of rat cerebella to ethanol under hypoxia imposedin vitro

This publication has 33 references indexed in Scilit:

• The Significance of Family History Status in Relation to Neuropsychological Test Performance and Cerebral Glucose Metabolism Studied with Positron Emission Tomography in Older Alcoholic Patients
  Alcohol, Clinical and Experimental Research, 1998
• Acute exposure to alcohol during early postnatal life causes a deficit in the total number of cerebellar Purkinje cells in the rat
  Journal of Comparative Neurology, 1995
• Cleavage of DNA to Large Kilobase Pair Fragments Occurs in Some Forms of Necrosis as Well as Apoptosis
  Biochemical and Biophysical Research Communications, 1995
• Fetal alcohol syndrome: the vulnerability of the developing brain and possible mechanisms of damage
  Metabolic Brain Disease, 1994
• Apoptosis: Molecular Control Point in Toxicity
  Toxicology and Applied Pharmacology, 1994
• In Vitro Hypoxia Induces Expression of the NR2C Subunit of the NMDA Receptor in Rat Cortex and Hippocampus
  Journal of Neurochemistry, 1994
• The Effects of the Timing of Ethanol Exposure during the Brain Growth Spurt on the Number of Cerebellar Purkinje and Granule Cell Nuclear Profiles
  Alcohol, Clinical and Experimental Research, 1993
• Guidelines for Use of Terminology Describing the Impact of Prenatal Alcohol on the Offspring
  Alcohol, Clinical and Experimental Research, 1989
• A CHANGE IN SUSCEPTIBILITY OF RAT CEREBELLAR PURKINJE CELLS TO DAMAGE BY ALCOHOL DURING FETAL, NEONATAL AND ADULT LIFE
  Neuropathology and Applied Neurobiology, 1982
• Blood ethanol levels in sober alcohol users seen in an emergency room
  Life Sciences, 1981