Biochemical Staging of Synucleinopathy and Amyloid Deposition in Dementia With Lewy Bodies
Open Access
- 1 March 2006
- journal article
- research article
- Published by Oxford University Press (OUP) in Journal of Neuropathology and Experimental Neurology
- Vol. 65 (3) , 278-288
- https://doi.org/10.1097/01.jnen.0000205145.54457.ea
Abstract
The primary feature of dementia with Lewy bodies (DLB) is the aggregation of alpha-synuclein into characteristic lesions: Lewy bodies (LBs) and LewyKeywords
This publication has 45 references indexed in Scilit:
- Dementia in Parkinson's disease: cause and treatmentCurrent Opinion in Neurology, 2004
- Dementia with Lewy bodiesThe Lancet Neurology, 2004
- Alpha-synuclein and neurodegenerative diseasesNature Reviews Neuroscience, 2001
- Report of the second dementia with Lewy body international workshopNeurology, 1999
- α-Synuclein in filamentous inclusions of Lewy bodies from Parkinson’s disease and dementia with Lewy bodiesProceedings of the National Academy of Sciences, 1998
- Consensus guidelines for the clinical and pathologic diagnosis of dementia with Lewy bodies (DLB)Neurology, 1996
- Plaque-Only Alzheimer Disease is Usually the Lewy Body Variant, and Vice VersaJournal of Neuropathology and Experimental Neurology, 1993
- Senile dementia of Lewy body typeJournal of the Neurological Sciences, 1990
- The Lewy body variant of Alzheimer's diseaseNeurology, 1990
- CLINICAL AND PATHOLOGICAL FEATURES OF DIFFUSE CORTICAL LEWY BODY DISEASE (LEWY BODY DEMENTIA)Brain, 1987