LEFT-VENTRICULAR OVERLOAD CAUSED BY COLLATERAL PULMONARY CIRCULATION IN CHRONIC PNEUMOPATHIES - MYTH OR REALITY

  • 1 January 1981
    • journal article
    • research article
    • Vol. 23  (2) , 121-125
Abstract
The collateral pulmonary blood flow was measured by the dye dilution method in 4 patients with chronic obstructive pulmonary disease, in 16 patients with diffuse pulmonary fibrosis, in 15 patients with pulmonary embolism, and in 3 patients with primary pulmonary hypertension. Collateral pulmonary circulation was small in the first 2 groups (0.3% and 1.7% of the pulmonary blood flow, respectively), absent in primary pulmonary hypertension, and high (14.0% of pulmonary flow) in pulmonary embolism. The magnitude of the bronchopulmonary collateral circulation did not correlate with the degree of precapillary pulmonary hypertension, left atrial pressure and left ventricular enddiastolic pressure. The bronchopulmonary collateral flow apparently does not produce diastolic left ventricular overload in chronic pneumopathies.

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