Genetic variants at the 9p21 locus contribute to atherosclerosis through modulation of ANRIL and CDKN2A/B

This publication has 33 references indexed in Scilit:

• Long non-coding RNA ANRIL is required for the PRC2 recruitment to and silencing of p15INK4B tumor suppressor gene
  Oncogene, 2010
• Associations of 9p21 variants with cutaneous malignant melanoma, nevi, and pigmentation phenotypes in melanoma-prone families with and without CDKN2A mutations
  Familial Cancer, 2010
• Molecular Interplay of the Noncoding RNA ANRIL and Methylated Histone H3 Lysine 27 by Polycomb CBX7 in Transcriptional Silencing of INK4a
  Published by Elsevier ,2010
• Variation in CDKN2A at 9p21.3 influences childhood acute lymphoblastic leukemia risk
  Nature Genetics, 2010
• Genome-wide association study identifies variants at 9p21 and 22q13 associated with development of cutaneous nevi
  Nature Genetics, 2009
• Sequence variants on chromosome 9p21.3 confer risk for atherosclerotic stroke
  Annals of Neurology, 2009
• The same sequence variant on 9p21 associates with myocardial infarction, abdominal aortic aneurysm and intracranial aneurysm
  Nature Genetics, 2008
• Susceptibility to coronary artery disease and diabetes is encoded by distinct, tightly linked SNPs in the ANRIL locus on chromosome 9p
  Human Molecular Genetics, 2007
• Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls
  Nature, 2007
• Regulation of the INK4b–ARF–INK4a tumour suppressor locus: all for one or one for all
  Nature Reviews Molecular Cell Biology, 2006