Ethanol withdrawal is associated with increased extracellular glutamate in the rat striatum
- 1 September 1995
- journal article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 283 (1-3) , 177-183
- https://doi.org/10.1016/0014-2999(95)00344-k
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Extracellular Glutamate Is Increased in Thalamus During Thiamine Deficiency‐Induced Lesions and Is Blocked by MK‐801Journal of Neurochemistry, 1993
- Cerebral Vulnerability Is Associated with Selective Increase in Extracellular Glutamate Concentration in Experimental Thiamine DeficiencyJournal of Neurochemistry, 1993
- Chronic Ethanol Intoxication Induces Differential Effects on GABAA and NMDA Receptor Function in the Rat BrainAlcohol, Clinical and Experimental Research, 1993
- Ethanol Inhibits NMDA Receptor‐Mediated Excitotoxicity in Rat Primary Neuronal CulturesAlcohol, Clinical and Experimental Research, 1993
- The Involvement of NMDA Receptors in Acute and Chronic Effects of EthanolAlcohol, Clinical and Experimental Research, 1992
- Management of the Alcohol withdrawal SyndromeAnnual Review of Medicine, 1991
- Ethanol inhibits NMDA-induced increases in free intracellular Ca2+ in dissociated brain cellsBrain Research, 1989
- Ethanol Inhibits NMDA-Activated Ion Current in Hippocampal NeuronsScience, 1989
- Alterations in γ‐Aminobutyric Acid Receptor Sensitivity Following Acute and Chronic Ethanol TreatmentsJournal of Neurochemistry, 1980
- Dissociation of alcohol tolerance and dependenceNature, 1976