Mismatch Repair Deficiency Interferes with the Accumulation of Mutations in Chronically Stimulated B Cells and Not with the Hypermutation Process
- 1 July 1998
- Vol. 9 (1) , 127-134
- https://doi.org/10.1016/s1074-7613(00)80594-4
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Mismatch Repair Co-opted by HypermutationScience, 1998
- Cells strongly expressing Ig(kappa) transgenes show clonal recruitment of hypermutation: a role for both MAR and the enhancers.The EMBO Journal, 1997
- MutS homologs in mammalian cellsCurrent Opinion in Genetics & Development, 1997
- Involvement of mouse Mlh1 in DNA mismatch repair and meiotic crossing overNature Genetics, 1996
- Meiotic Pachytene Arrest in MLH1-Deficient MiceCell, 1996
- Inactivation of the mouse Msh2 gene results in mismatch repair deficiency, methylation tolerance, hyperrecombination, and predisposition to cancerCell, 1995
- Male mice defective in the DNA mismatch repair gene PMS2 exhibit abnormal chromosome synapsis in meiosisCell, 1995
- Age-related decrease in the proportion of germinal center B cells from mouse Peyer's patches is accompanied by an accumulation of somatic mutations in their immunoglobulin genesEuropean Journal of Immunology, 1994
- Elements regulating somatic hypermutation of an immunoglobulin κ gene: Critical role for the intron enhancer/matrix attachment regionCell, 1994
- Origin of Antibody VariationNature, 1966