Induction of Bone Morphogenetic Protein-6 in Skin Wounds. Delayed Reepitheliazation and Scar Formation in BMP-6 Overexpressing Transgenic Mice
- 1 December 1998
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 111 (6) , 1145-1152
- https://doi.org/10.1046/j.1523-1747.1998.00407.x
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Overexpression of bone morphogenetic protein-6 (BMP-6) in the epidermis of transgenic mice: inhibition or stimulation of proliferation depending on the pattern of transgene expression and formation of psoriatic lesions.The Journal of cell biology, 1996
- Expression and growth inhibitory effect of decapentaplegic Vg-related protein 6: evidence for a regulatory role in keratinocyte differentiation.Proceedings of the National Academy of Sciences, 1994
- One systemic administration of transforming growth factor-beta 1 reverses age- or glucocorticoid-impaired wound healing.Journal of Clinical Investigation, 1993
- Growth factors in development, transformation, and tumorigenesisCell, 1991
- Identification of transforming growth factor beta family members present in bone-inductive protein purified from bovine bone.Proceedings of the National Academy of Sciences, 1990
- Changes in keratinocyte adhesion during terminal differentiation: Reduction in fibronectin binding precedes α5β1 integrin loss from the cell surfaceCell, 1990
- Cell migration is essential for sustained growth of keratinocyte colonies: The roles of transforming growth factor-α and epidermal growth factorCell, 1987
- Tenascin: an extracellular matrix protein involved in tissue interactions during fetal development and oncogenesisCell, 1986
- Cutaneous tissue repair: Basic biologic considerations. IJournal of the American Academy of Dermatology, 1985
- Accelerated wound repair, cell proliferation, and collagen accumulation are produced by a cartilage-derived growth factor.The Journal of cell biology, 1985