Role of the transcription factor AML-1 in acute leukemia and hematopoietic differentiation
- 10 March 2000
- Vol. 245 (2) , 223-235
- https://doi.org/10.1016/s0378-1119(00)00014-7
Abstract
No abstract availableKeywords
This publication has 91 references indexed in Scilit:
- TLE, the Human Homolog of Groucho, Interacts with AML1 and Acts as a Repressor of AML1-Induced TransactivationBiochemical and Biophysical Research Communications, 1998
- Histone acetylation: A possible mechanism for the inheritance of cell memory at mitosisBioEssays, 1997
- A transcriptional co-repressor that interacts with nuclear hormone receptorsNature, 1995
- Ligand-independent repression by the thyroid hormone receptor mediated by a nuclear receptor co-repressorNature, 1995
- Requirement of Transcription Factor PU.1 in the Development of Multiple Hematopoietic LineagesScience, 1994
- Inhibition of DNA replication factor RPA by p53Nature, 1993
- The acidic transcriptional activation domains of VP16 and p53 bind the cellular replication protein A and stimulate in vitro BPV-1 DNA replicationCell, 1993
- The transactivator proteins VP16 and GAL4 bind replication factor ACell, 1993
- Transcriptionally active chimeric gene derived from the fusion of the AML1 gene and a novel gene on chromosome 8 in t(8;21) leukemic cellsCancer Genetics and Cytogenetics, 1992
- Premature expression of the macrophage colony-stimulating factor receptor on a multipotential stem cell line does not alter differentiation lineages controlled by stromal cells used for coculture.The Journal of Experimental Medicine, 1991