Tumor surveillance via the ARF–p53 pathway
Open Access
- 1 October 1998
- journal article
- review article
- Published by Cold Spring Harbor Laboratory in Genes & Development
- Vol. 12 (19) , 2984-2991
- https://doi.org/10.1101/gad.12.19.2984
Abstract
A biweekly scientific journal publishing high-quality research in molecular biology and genetics, cancer biology, biochemistry, and related fieldsThis publication has 71 references indexed in Scilit:
- Regulation of p53 stability by Mdm2Nature, 1997
- Mdm2 promotes the rapid degradation of p53Nature, 1997
- Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4aCell, 1997
- Cancer Cell CyclesScience, 1996
- Homozygotes for CDKN2 (p16) germline mutation in Dutch familial melanoma kindredsNature Genetics, 1995
- A Cell Cycle Regulator Potentially Involved in Genesis of Many Tumor TypesScience, 1994
- A new regulatory motif in cell-cycle control causing specific inhibition of cyclin D/CDK4Nature, 1993
- WAF1, a potential mediator of p53 tumor suppressionCell, 1993
- Altered cell cycle arrest and gene amplification potential accompany loss of wild-type p53Published by Elsevier ,1992
- In vitro establishment is not a sufficient prerequisite for transformation by activated ras oncogenesCell, 1986