Th1- and Th2-cell commitment during infectious disease: asymmetry in divergent pathways
- 1 August 2001
- journal article
- review article
- Published by Elsevier in Trends in Immunology
- Vol. 22 (8) , 450-457
- https://doi.org/10.1016/s1471-4906(01)01975-5
Abstract
No abstract availableKeywords
This publication has 87 references indexed in Scilit:
- Interleukin-18 (IL-18) Enhances Innate IL-12-Mediated Resistance toToxoplasma gondiiInfection and Immunity, 2000
- Stat6 activation and Th2 cell proliferation driven by CD28 signalsEuropean Journal of Immunology, 2000
- Signaling and Transcription in T Helper DevelopmentAnnual Review of Immunology, 2000
- Host Defense Mechanisms Triggered by Microbial Lipoproteins Through Toll-Like ReceptorsScience, 1999
- Two Roads Diverged: Interferon α/β– and Interleukin 12–mediated Pathways in Promoting T Cell Interferon γ Responses during Viral InfectionThe Journal of Experimental Medicine, 1999
- Parasite dose determines the Th1/Th2 nature of the response toLeishmania major independently of infection route and strain of host or parasiteEuropean Journal of Immunology, 1998
- INDUCTION OF TH1 AND TH2 CD4+T CELL RESPONSES:The Alternative ApproachesAnnual Review of Immunology, 1997
- CD28/B7 SYSTEM OF T CELL COSTIMULATIONAnnual Review of Immunology, 1996
- CD40 Ligand Is Required for Protective Cell-Mediated Immunity to Leishmania majorImmunity, 1996
- Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4Immunity, 1995