Increased Nitration of Sarcoplasmic Reticulum Ca 2+ -ATPase in Human Heart Failure
- 1 March 2005
- journal article
- research article
- Published by Wolters Kluwer Health in Circulation
- Vol. 111 (8) , 988-995
- https://doi.org/10.1161/01.cir.0000156461.81529.d7
Abstract
Background— Reduced sarcoplasmic reticulum (SR) Ca2+-ATPase (SERCA2a isoform) activity is a major determinant of reduced contractility in heart failure. Ca2+-ATPase inactivation can occur through S...Keywords
This publication has 37 references indexed in Scilit:
- Functional nitric oxide synthase mislocalization in cardiomyopathyJournal of Molecular and Cellular Cardiology, 2004
- Abnormal frequency-dependent responses represent the pathophysiologic signature of contractile failure in human myocardiumJournal of Molecular and Cellular Cardiology, 2004
- Peroxynitrite-induced cardiac depression: role of myofilament desensitization and cGMP pathwayCardiovascular Research, 2003
- Up‐regulation of cardiac nitric oxide synthase 1‐derived nitric oxide after myocardial infarction in senescent ratsThe FASEB Journal, 2003
- Phospholamban: a crucial regulator of cardiac contractilityNature Reviews Molecular Cell Biology, 2003
- Gender Influences on Sarcoplasmic Reticulum Ca2+-handling in Failing Human MyocardiumJournal of Molecular and Cellular Cardiology, 2001
- Transmembrane Helix M6 in Sarco(endo)plasmic Reticulum Ca2+-ATPase Forms a Functional Interaction Site with PhospholambanJournal of Biological Chemistry, 1999
- Inducible Nitric Oxide Synthase (iNOS) in the Human Heart: Expression and Localization in Congestive Heart FailureJournal of Molecular and Cellular Cardiology, 1998
- Myocardial calcium-independent nitric oxide synthase activity is present in dilated cardiomyopathy, myocarditis, and postpartum cardiomyopathy but not in ischaemic or valvar heart disease.Heart, 1995
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976