Inhibition of protein kinase A and cyclic AMP response element (CRE)-specific transcription factor binding by Δ9-tetrahydrocannabinol (Δ9-THC): A putative mechanism of cannabinoid-induced immune modulation
- 15 May 1997
- journal article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 53 (10) , 1477-1484
- https://doi.org/10.1016/s0006-2952(97)82441-0
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Suppression of the humoral immune response by cannabinoids is partially mediated through inhibition of adenylate cyclase by a pertussis toxin-sensitive G-protein coupled mechanismBiochemical Pharmacology, 1994
- Flow‐cytometric detection of the RIα subunit of type I cAMP‐dependent protein kinase in human cellsCytometry, 1994
- Δ9-tetrahydrocannabinol selectivity inhibits T-cell dependent humoral immune responses through direct inhibition of accessory T-cell functionImmunopharmacology, 1993
- Molecular characterization of a peripheral receptor for cannabinoidsNature, 1993
- Inhibition of adenylate cyclase by Δ9-Tetrahydrocannabinol in mouse spleen cells: A potential mechanism for cannabinoid- mediated immunosuppressionLife Sciences, 1992
- Marijuana components suppress induction and cytolytic function of murine cytotoxic T cells in vitro and in vivoJournal of Toxicology and Environmental Health, 1991
- Structure of a cannabinoid receptor and functional expression of the cloned cDNANature, 1990
- In vitro and in vivo suppressive effects of delta-9-tetrahydrocannabinol on interferon production by murine spleen cellsInternational Journal of Immunopharmacology, 1986
- Cyclic adenosine 3′,5′-monophosphate in human lymphocytes. Alterations after phytohemagglutinin stimulationJournal of Clinical Investigation, 1971
- Marihuana ChemistryScience, 1970