Energy metabolism at high altitude (3,475 m)

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Abstract
Maximal work capacity (Vo2) on the bicycle ergometer was decreased in three groups of men, one group acclimated to sea level, and two groups acclimated to 1,610 m. At 3,475 m, maximal Vo2 in milliliters per kilogram body weight per minute was reduced by 17% for the sea level group, and by 10% for the group from 1,610 m. Although there was a difference of approximately 7% in Vo2 between sea level and 1,610 m, there was no measurable beneficial effect of acclimatization at 1,610 m in improving maximal work at 3,475 m. Maximal work capacity and maximal V02 did not improve over a 20-day period at altitude. VE STPD [pulmonary minute ventilation at standard temperature and pressure, dry air] was decreased, and VE BTPS [pulmonary minute ventilation at body temperature and pressure, saturated with water vapor] increased on arrival at altitude with a gradual increase in both during prolonged exposure. Pulse rates at rest, and moderate exercise, were consistently high at high altitudes, whereas the maximal pulse rates gradually declined. Oxygen consumption at the basal, sitting rest, and moderate exercise states was not markedly changed by altitude. The physiological cause for the cessation of maximal work at altitude remains obscure. Under the conditions of this study, a) the 1,610-m elevation did not seem to be beneficial in improving the maximal work at 3,475 m, b) a 20-day acclimatization period at 3,475 m did not result in a superior submaximal or maximal work performance on return to sea level, and c) individuals can adequately perform submaximal work even after the initial high-altitude exposure.

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