β-Glucuronidase Is an Optimal Normalization Control Gene for Molecular Monitoring of Chronic Myelogenous Leukemia
- 31 July 2006
- journal article
- research article
- Published by Elsevier in The Journal of Molecular Diagnostics
- Vol. 8 (3) , 385-389
- https://doi.org/10.2353/jmoldx.2006.050150
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Molecular Monitoring of Chronic Myelogenous LeukemiaThe Journal of Molecular Diagnostics, 2006
- Phase I/II trial of adding semisynthetic homoharringtonine in chronic myeloid leukemia patients who have achieved partial or complete cytogenetic response on imatinibCancer, 2005
- Monitoring patients in complete cytogenetic remission after treatment of CML in chronic phase with imatinib: patterns of residual leukaemia and prognostic factors for cytogenetic relapseLeukemia, 2005
- Clinical Pharmacokinetics of ImatinibClinical Pharmacokinetics, 2005
- Loss of Response to Imatinib: Mechanisms and ManagementHematology-American Society Hematology Education Program, 2005
- Real-time quantitative PCR analysis can be used as a primary screen to identify patients with CML treated with imatinib who have BCR-ABL kinase domain mutationsBlood, 2004
- Distinct roles of c-Abl and Atm in oxidative stress response are mediated by protein kinase C δGenes & Development, 2004
- Differential stability of control gene and fusion gene transcripts over time may hamper accurate quantification of minimal residual disease – a study within the Europe Against Cancer ProgramLeukemia, 2004
- Standardization and quality control studies of ‘real-time’ quantitative reverse transcriptase polymerase chain reaction of fusion gene transcripts for residual disease detection in leukemia – A Europe Against Cancer ProgramLeukemia, 2003
- Quantitative real‐time reverse‐transcription polymerase chain reaction for diagnosis of BCR‐ABL positive leukemias and molecular monitoring following allogeneic stem cell transplantationEuropean Journal of Haematology, 2003