Cardiac Hypertrophy and Impaired Relaxation in Transgenic Mice Overexpressing Triadin 1
Open Access
- 1 February 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (6) , 4142-4149
- https://doi.org/10.1074/jbc.m006443200
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Localization and Characterization of the Calsequestrin-binding Domain of Triadin 1Journal of Biological Chemistry, 2000
- Dual Regulation of the Skeletal Muscle Ryanodine Receptor by Triadin and CalsequestrinBiochemistry, 1998
- Regulation of Ca2+ signaling in transgenic mouse cardiac myocytes overexpressing calsequestrin.Journal of Clinical Investigation, 1998
- A Proteasome Activator Subunit Binds CalciumJournal of Biological Chemistry, 1995
- Polar zippers: Their role in human diseaseProtein Science, 1994
- KEKE motifsFEBS Letters, 1994
- Localization and partial characterization of the oligomeric disulfide-linked molecular weight 95,000 protein (triadin) which binds the ryanodine and dihydropyridine receptors in skeletal muscle triadic vesiclesBiochemistry, 1991
- Stretch-Activated Channels in Heart CellsJournal of Cardiovascular Pharmacology, 1991
- Isolation of a terminal cisterna protein which may link the dihydropyridine receptor to the junctional foot protein in skeletal muscleBiochemistry, 1990
- Biochemical Mechanisms of Cardiac HypertrophyAnnual Review of Physiology, 1987