A highly conserved tyrosine residue at codon 845 within the kinase domain is not required for the transforming activity of human epidermal growth factor receptor
- 1 July 1992
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 186 (2) , 768-774
- https://doi.org/10.1016/0006-291x(92)90812-y
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
- Foundation for Promotion of Cancer Research
This publication has 21 references indexed in Scilit:
- Signal transduction by receptors with tyrosine kinase activityPublished by Elsevier ,1990
- Site-directed mutagenesis by overlap extension using the polymerase chain reactionGene, 1989
- The Protein Kinase Family: Conserved Features and Deduced Phylogeny of the Catalytic DomainsScience, 1988
- GROWTH FACTOR RECEPTOR TYROSINE KINASESAnnual Review of Biochemistry, 1988
- Epidermal-Growth-Factor-Dependent Transformation by a Human EGF Receptor Proto-OncogeneScience, 1987
- Overexpression of the human EGF receptor confers an EGF-dependent transformed phenotype to NIH 3T3 cellsCell, 1987
- Tyrosine phosphorylation regulates the biochemical and biological properties of pp60c-srcCell, 1987
- Activation and suppression of pp60c-src transforming ability by mutation of its primary sites of tyrosine phosphorylationCell, 1987
- Replacement of insulin receptor tyrosine residues 1162 and 1163 compromises insulin-stimulated kinase activity and uptake of 2-deoxyglucoseCell, 1986
- Mutagenesis of Fujinami Sarcoma Virus: Evidence that tyrosine phosphorylation of P130gag-fps modulates its biological activityCell, 1984